Burst fracture

A burst fracture is a type of compression fracture which results in distruption of the posterior vertebral body cortex with retropulsion into the spinal canal. When in the thoraco lumbar level it tends to occur between T9 and L5 levels ³.  Burst fractures may be stable or unstable.

Pathology

Mechanism

It is a result of a compressive high energy injury (axial loading), much like the Jefferson fracture.  Typically it occurs following a fall, landing on the feet, from a significant height.

The intervertebral disc is driven into the vertebral body below.

All patients require a CT to assess the injury and evaluate the extent of retropulsed fragments which may enter the spinal canal.

Radiographic features

General features include

• 'burst' vertebral body on axial CT.
• loss of posterior vertebral height on lateral views.
• retropulsed fragments in the spinal canal.
• interpedicular widening

Cardiogenic and Non-cardiogenic
Pulmonary Edema

General Considerations Increase in the fluid in the lung Generally, divided into cardiogenic and non-cardiogenic categories. Congestive heart failure is the leading diagnosis in hospitalized patients older than 65 Pathophysiology Fluid first accumulates in and around the capillaries in the interlobular septa (typically at a wedge pressure of about 15 mm Hg) Further accumulation occurs in the interstitial tissues of the lungs Finally, with increasing fluid, the alveoli fill with edema fluid (typically wedge pressure is 25 mm Hg or more) Causes Cardiogenic pulmonary edema. Heart failure Coronary artery disease with left ventricular failure. Cardiac arrhythmias Fluid overload -- for example, kidney failure. Cardiomyopathy Obstructing valvular lesions -- for example, mitral stenosis  Myocarditis and infectious endocarditis Non-cardiogenic pulmonary edema -- due to changes in capillary permeability Smoke inhalation. Head trauma Overwhelming sepsis. Hypovolemia shock Re-expansion  By drainage of a large pleural effusion with thoracentesis Of the lung collapsed by a large pneumothorax High altitude pulmonary edema Disseminated intravascular coagulopathy (DIC) Near-drowning Overwhelming aspiration  Heroin overdose Adult (acute) respiratory distress (deficiency) syndrome (ARDS)  Clinical syndrome consisting of Pulmonary edema associated with severe respiratory distress Cyanosis refractory to oxygen administration Decreased lung compliance Lower pulmonary capillary wedge pressure (PCW < 18mm Hg) than cardiogenic pulmonary edema Most patients who survive have normal-appearing lungs Some patients develop pulmonary fibrosis Clinical Findings Shortness of breath Hemoptysis Orthopnea Dyspnea on exertion Cough, wheezing Anxiety and restlessness Cyanosis Imaging Findings Radiographic findings can lag behind physiologic changes The key findings of cardiogenic pulmonary edema Kerley B lines (septal lines) Seen at the lung bases, usually no more than 1 mm thick and 1 cm long, perpendicular to the pleural surface Pleural effusions Usually bilateral, frequently the right side being larger than the left If unilateral, more often on the right Fluid in the fissures Thickening of the major or minor fissure Peribronchial cuffing Visualization of small doughnut-shaped rings representing fluid in thickened bronchial walls Collectively, the above four findings comprise pulmonary interstitial edema The heart may or may not be enlarged When the fluid  enters the alveoli themselves, the airspace disease is typically diffuse, and there are no air bronchograms Non-cardiogenic pulmonary edema Bilateral, peripheral air space disease with air bronchograms or central bat-wing pattern Kerley B lines and pleural effusions are uncommon Typically occurs 48 hours or more after the initial insult Stabilizes at around five days and may take weeks to completely clear On CT Gravity-dependent consolidation or ground glass opacification Air bronchograms are common Differential Diagnosis Diffuse pneumonia Massive aspiration  Pulmonary hemorrhage  Treatment Cardiogenic pulmonary edema and non-cardiogenic pulmonary edema, with the exception of ARDS, can resolve within hours to several days Cardiogenic pulmonary edema is usually treated with a combination of Oxygen Diuretics Lasix, etc. Nitrates Nitroglycerin, etc. Natriuretic peptides Nesiritide, etc. Morphine Inotropic agents Dopamine, dobutamine, digoxin, etc. Angiotensin converting enzyme (ACE) inhibitors Beta-blockers Carvedilol, etc. For non-cardiogenic pulmonary edema, the predisposing condition should be treated Treatment is supportive Ventilator management. Antibiotic therapy, when necessary Corticosteroids

Tuberculose Pulmonaire


Cliché thoracique de face et de profil: infiltrat micronodulaire apical gauche, avec doute sur la présence d'une image de caverne en rétro claviculaire. Pas d'anomalie retenue de profil.

TDM: confirme l'atteinte typique, bilatérale, avec présence de cavernes au niveau apical gauche. Confirmation biologique du diagnostic. 

Osteochondroma

General Considerations Osteochondroma is the most common benign bone tumor and the most common skeletal neoplasm They account for approximately 35% of all benign bone tumors and 9% of all bone tumors They are cartilage-capped bony projections from the external surface of a bone They occur only in bones which form by enchondral bone formation and most commonly found around the knee (40%) and shoulder, although they can occur in any bone Almost all are diagnosed in patients under the age of 20 with a marked male:female predominance (3:1) They grow until skeletal maturity and then stop growing when the epiphyseal plate fuses, although the cartilage cap can continue to grow slowly until about age 30 Osteochondromas tend to occur near an epiphyseal growth plate and grow away from the physis Clinical Findings Most are diagnosed incidentally Or, they may come to clinical attention because they produce a mass They are usually asymptomatic When painful, they should be evaluated for Mechanical irritation and inflammation of the surrounding soft tissues Fracture of the stalk Avascular necrosis of the cartilaginous cap Malignant degeneration Imaging Findings Conventional radiography is the study of first choice CT can be used to determine if the marrow and cortices of the lesion are continuous with the parent bone MRI can be used to asses surrounding soft tissues and to measure the thickness of the cartilage cap, which can be important in evaluating for malignant generation A thick cartilaginous cap (>1 cm) in adults should raise the possibility of malignant transformation They can vary is size considerably, with the average tubular bone lesion being about 4 cm Osteochondromas can either be sessile (flat) or pedunculated (stalk) Sessile lesions are more likely to be associated with abnormalities of tubulation of the underlying bone leading to metaphyseal widening or a "trumpet shaped” deformity on x-ray Malignant Degeneration Fewer than 1% of solitary osteochondromas undergo malignant degeneration of the cartilage cap into secondary chondrosarcoma It is usually preceded by New onset of growth of the lesion Rapid growth of a lesion, or New onset of pain The risk of malignant degeneration increases with an increase in the number and size of the osteochondromas In general, a sessile lesion is more likely to degenerate into sarcoma than a pedunculated lesion (exostosis) Associations and Syndromes Hereditary multiple exostoses Autosomal dominant condition Short stature Multiple osteochondromas Asymmetric growth at the knees and ankles Risk of malignant degeneration is 1-20% Dysplasia epiphysealis hemimelica (DEH, Trevor disease) Osteochondromas arising in the epiphyses Involve the joint Lesions restricted to one side of the body–either left or right May be multiple lesions in a single limb Primarily involves one side of an epiphysis Medial side is affected twice as often as the lateral side Usually occurs in infants or young children Treatment There is no treatment necessary for asymptomatic osteochondromas The cornerstone of treatment is observation because most lesions are asymptomatic If the lesion is causing pain or neurologic symptoms due to compression, it should be resected at the base None of the cartilage cap or perichondrium should be left in the resection bed or  recurrence can occur. As long as the entire cartilage cap is removed there should be no recurrence Patients with many large osteochondromas should have regular radiographic screening exams for the early detection of malignant transformation

- See more at: http://www.learningradiology.com/archives2012/COW%20532-Osteochondroma/osteochondromacorrect.html#sthash.uWETvknz.dpuf

FIBROSE RETROPERITONEALE BENIGNE DE TYPE IDIOPATHIQUE CONFIRMEE PAR INTERVENTION CHIRURGICALE

Cette urographie met en évidence un aspect particulier de fibrose rétropéritonéale avec d'une part une localisation unilatérale et d'autre part l'absence d'attraction vers la ligne médiane de l'uretère.

UIV : perméabilité des voies urinaires gauches. A droite, syndrome obstructif modéré avec néphrographie intense et dilatation modérée des cavités pyélo-calicielles et de l'artère proximale. Ce syndrome obstructif est secondaire à une sténose localisée en regard de L3 centrée et régulière (flèche). Il n'y a pas d'attraction évidente vers la ligne médiane.

KYSTE JUXTA-ARTICULAIRE DE LA CHEVILLE(GANGIAL CYST)

La tomographie confirme l'aspect de la lésion kystique et montre une communication avec l'interligne articulaire (flèche).

Le kyste juxta-articulaire est une lésion kystique bénigne composé d'un tissu fibreux à évolution mucoïde , localisé à l'os sous-chondral. L'articulation voisine doit être normale. C'est en fait un diagnostic d'élimination. Cliniquement il est asymptomatique ou associé à quelques douleurs mécaniques. Autres sièges moins fréquents : extrémité supérieure du tibia, tête cubitale,olécrâne.

Diagnostic : KYSTE HYDATIQUE PLEIN DU LOBE INFERIEUR DROIT DE 15 cm DE DIAMETRE.

Les calcifications du kyste hydatique au niveau du poumon sont exceptionnelles et infraradiologiques. La TDM a permis demettre en évidence de petitescalcifications pariétales dans 2 cas sur36 explorés par TDM.

             RADIOGRAPHIE DIGITALISEE DU THORAX deface en décubitus : importante opacitédes deux tiers inférieurs de l'hémithorax.                                                                             

TDM THORACIQUE : masse de densité liquidienne (1 U.H., de 10,5 cm de diamètre), homogène, tassement pulmonaire antérieur.